Basicão: Durateston + Hemogenin

[contaalta 0]

Fla ae pessoal, sou novo aquí por isso antes de começar... saudações a todos!!

Não sou novato com uso de roids, porém ja faz 7 anos desde o ultimo ciclo. Por causa de trabalho, mudanças e etc. Fiquei muito tempo parado e perdi muito do que havia ganho com otros ciclos.

Eu tava pensando em fazer um ciclo para dar um up na musculatura somente com:

Hemogenin (1 comp ao dia x 6 sem)

Durateston (750mg x 8 sem)

Silimarina (300mg) para proteger o fígado

Whey a vontade

Falo de Dura e Hemo, porque foi as que usei em outros 2 ciclos e me deram um ótimo resultado, é mais facil de encontrar mercadoria original e o custo naum é dos maiores.. Porém as outras vezes fiz com Deca (200 mg semana)...

Alguém já tomou somente Hemo + Dura, da um levante legal ou vai passar quase despercebido??

Opinem.

Há Braços.

[emilio1709 0]

troca esse hemogenim por outra droga... manda um ciclo6 no lugar... hemogenim fode com o figado...

[Gusmão420 230]

Tb acho q talvez 6sem de hemo seria mto.

Silimarina 2 sem antes do ciclo e/ou 2sem após o fim da TPC.

Abraço

[Filipesep 20]

Manolo, pq ao inves de hemo vc nao manda diana? Na minha opiniao, e creio q da maioria, diana eh beeeem melhor.

Abracos

[Kracks 80]

mano eu so a favor de trocar esse hemo por diana tbm, no resto nao tenhu nada a contrariar

[diggoskt 37]

a dosagem de dura ta ok

mas nao joga seu figado fora por causa de dinheiro

manda um diana :X

caso vc nao tenha acesso ao diana ainda acho uma melhor alternativa m drol

[Ferret 46]

que exagero o pessoal pensar so pq o cara vai mandar oximetolona ele vai jogar o figado fora, vai ter um cancer imediato, nao é assim tbm pessoal

dano no figado é o efeito colateral mais sensacionalista, mas o pior colateral é acreditar em tudo o que voce lê, esse forum é cheio de "bro science" e nego que acha que entende das coisas

quem quiser poe no google tradutor ae, preguiça de traduzir

2. Steroid Effects and Liver Damage

Liver damage is probably the most sensationalized of all side effects possible from steroid use. The media often focuses on this particular problem as if it occurs with every steroid, and in every person who takes them. Nothing could be further than the truth. Most anabolic steroids which are ingested orally pass through the liver, which functions as the body´s filtration system. When something goes through the liver, it is broken down by various enzymes, and passed along into the bloodstream. Most research on orally administered anabolic steroids focus on the fact that liver enzymes are elevated following ingestion. But does this necessarily mean that the liver is being damaged, does it? Of course not. Commonly, studies that focus on steroid toxicity often use absurd doses, or incorrectly focus on liver activity instead of damage. The liver functions as the filter for the human body.. it´s going to be activated whenever something (not just a steroid) passes through it. Does that show that steroids damage the liver? Let´s see what the scientists say..

There was an eight-week study done in 1999, which looked at the effects of an 8-week cycle of Oral steroids. The steroids examined were Halotestin (Fluoxymesterone), Dianabol (methylandrostanolone), or Winstrol (Stanozolol) on rats at the dose of 2mg/kg-body weight, administered five times a week for 8 weeks. That s almost 200mgs/day of any of those steroids, for a 200lb user. That is, I´ll speculate, much more than the average person would use on a cycle. In fact, I have never, in my years of researching steroids and speaking with athletes, heard of anybody using 200mgs/day of Halotestin, Winstrol, or Dianabol. Ever.

And, at the end of that study, In vivo, each rat still had liver enzyme levels that were within normal range!

(*Med Sci Sports Exerc. 1999 Feb;31(2):243-50, Rat liver lysosomal and mitochondrial activities are modified by anabolic-androgenic steroids. Molano F, Saborido A, Delgado J, Moran M, Megias A.)

In another study, 16 bodybuilders using steroids were compared to 12 bodybuilders who were not. Then the bodybuilders who had used steroids stopped taking them for three months, at which points, the researchers found that liver enzymes had returned to the same levels as the non users. After only 3 months!

(*Int J Sports Med 1996 Aug;17(6):429-33, Body composition, cardiovascular risk factors and liver function in long-term androgenic-anabolic steroids using bodybuilders three months after drug withdrawal. Hartgens F, Kuipers H, Wijnen JA, Keizer HA.)

We can see from the chart below that ex-steroid users have totally normal liver enzymes one year after they stop using& .in fact, for some liver enzymes, even the current users have normal scores!

(*Journal of Steroid Biochemistry and Molecular Biology. 84 (2003) 369-375)

Editado Junho 14, 2011 às 01:46 por Ferret

[drowning 52]

Concordo com o amigo Ferret, é muito terror em cima dessa droga.

Claro que é necessário saber usa-la.

Abraços

Editado Junho 14, 2011 às 02:13 por drowning

[Tycoon 101]

contribuindo também

Clin J Sport Med. 1999 Jan;9(1):34-9.

Anabolic steroid-induced hepatotoxicity: is it overstated?

Dickerman RD, Pertusi RM, Zachariah NY, Dufour DR, McConathy WJ.

Source

The Department of Biomedical Science, University of North Texas Health Science Center, Fort Worth 76107-2699, USA.

Abstract

OBJECTIVE:

There have been numerous reports of hepatic dysfunction secondary to anabolic steroid use based on elevated levels of serum aminotransferases. This study was conducted to distinguish between serum aminotransaminase elevations secondary to intense resistance training and anabolic steroid-induced hepatotoxicity in elite bodybuilders.

DESIGN:

This was a case-control study of serum chemistry profiles from bodybuilders using and not using anabolic steroids with comparisons to a cohort of medical students and patients with hepatitis.

PARTICIPANTS:

The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and nonexercising medical students (592) were used as controls.

MAIN OUTCOME MEASURES:

The focus in blood chemistry profiles was aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels.

RESULTS:

In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT.

CONCLUSION:

Prior reports of anabolic steroid-induced hepatotoxicity based on elevated aminotransferase levels may have been overstated, because no exercising subjects, including steroid users, demonstrated hepatic dysfunction based on GGT levels. Such reports may have misled the medical community to emphasize steroid-induced hepatotoxicity when interpreting elevated aminotransferase levels and disregard muscle damage. For these reasons, when evaluating hepatic function in cases of anabolic steroid therapy or abuse, CK and GGT levels should be considered in addition to ALT and AST levels as essential elements of the assessment.

PMID: 10336050 [PubMed - indexed for MEDLINE]

[Kracks 80]

minha indicação pela troca do hemo pelo diana eh mais pelos resultados do q pelo figueredo, as duas são bem parecidas em relação a hepatotoxicidade